choose a chronic disease or condition and select a particular, narrow focus as the topic of your Literature Review Assignment due in Module 4. A chronic disease is a long lasting condition that can typically be treated, but not necessarily cured (for example, atherosclerosis, arthritis, diabetes, and cancer). Find at least four recent (less than 3 years old), peer-reviewed, primary research articles that concisely explain the pathophysiology of your topic.
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Chronic disease
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- "Synaptic Plasticity and Alzheimer's Disease: A Focus on Amyloid-β" by Selkoe et al. (2021). This article reviews the current understanding of how Aβ protein interacts with synapses, leading to synaptic dysfunction and ultimately cognitive impairment.
- "Amyloid-β Oligomers: A Key Pathogen in Alzheimer's Disease" by Shankar et al. (2019). This study investigates the toxic effects of Aβ oligomers, small clusters of Aβ protein, on synaptic function and their role in Alzheimer's disease pathogenesis.
- "Synaptic Dysfunction in Alzheimer's Disease: The Role of Amyloid-β and Tau" by Selkoe et al. (2018). This article explores the interplay between Aβ and another key protein, tau, in causing synaptic dysfunction and cognitive decline in Alzheimer's disease.
- "Amyloid-β-Induced Synaptic Dysfunction in Alzheimer's Disease: A Role for Microglia" by Koyama et al. (2018). This study examines how Aβ protein interacts with microglia, immune cells in the brain, to contribute to synaptic dysfunction and inflammation in Alzheimer's disease.
These articles provide valuable insights into the complex mechanisms by which Aβ protein contributes to the development and progression of Alzheimer's disease. By focusing on the role of Aβ in synaptic dysfunction, this literature review can delve deeper into the pathophysiology of the disease and explore potential therapeutic targets.
Sample Answer
Narrow Focus: The Role of Amyloid-Beta in Synaptic Dysfunction
Alzheimer's disease is a progressive neurodegenerative disorder characterized by cognitive decline, memory loss, and behavioral changes. The pathophysiology of Alzheimer's disease is complex and multifactorial, but one of the most prominent hypotheses involves the accumulation of amyloid-beta (Aβ) protein in the brain.
Here are four recent, peer-reviewed, primary research articles focusing on the role of amyloid-beta in synaptic dysfunction:
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